Myosteatosis

A 12-week study on this protocol produced no measurable weight loss but reduced muscle loss by 15%, reflecting internal cellular repair that is invisible on a scale. Inner thigh fat is located inside the muscle fibers themselves — not beneath the skin — and is medically termed myosteatosis. Intramuscular fat must be burned locally by the muscle itself; there is no systemic transport mechanism to move it elsewhere. Intramuscular fat cannot be lost until the muscle is first rebuilt, because the damaged muscle lacks the capacity to burn the surrounding fuel. The root cause of myosteatosis is inactivity; all downstream effects — insulin resistance, mitochondrial decline, inflammation, scar tissue — develop on top of that. Insulin resistance develops within the muscle as myosteatosis progresses, preventing the muscle from effectively taking up fuel. Disrupted myokine metabolism driven by myosteatosis, SIBO, and nutritional deficiencies perpetuates muscle loss in a feedback loop. Muscle functions as a metabolic endocrine organ producing myokines including IL-6, IL-15, TNF-alpha, and myostatin. Myosteatosis is fat deposition within muscle tissue that reduces both muscle volume and contra…