Tumor and Cancer Treatment

Inducing senescence by lowering the non-stem proliferative lifespan slowed simulated tumor growth only transiently. When angiogenesis is inhibited by HBOT, tumors lose their nutrient supply and can shrink. Prolonged fasting is described as the single most effective strategy available for fighting cancer, including stage IV disease. The metabolic shift in cancer cells allows them to avoid normal cell death, enabling continuous growth and division. Tumor cells rely on glycolytic (sugar-based) metabolism rather than normal oxidative metabolism. Tumor and cancer cells have fundamentally altered mitochondrial metabolism, relying on glycolytic rather than normal oxidative metabolism. The metabolic effect of fasting starves cancer cells that rely on glucose and certain amino acids, outweighing the cost of muscle or weight loss. HBOT can cause mitochondria in tumor cells to rupture, destroying them. Cancer cells' metabolic shift allows them to avoid normal cell death, effectively living forever and continuously dividing. HBOT can cause mitochondria in tumor cells to rupture, destroying the tumor cells. Therapies that only increase senescence in non-stem cancer cells are unlikely to eradic…