Weight Gain

The antibiotic-dysbiosis-inflammation-insulin resistance pathway is a distinct mechanism of weight gain separate from sugar consumption or eating frequency. The dysbiosis-to-weight-gain pathway is a distinct metabolic route from the sugar-consumption or frequent-eating model of obesity. Fat storage allows the body to store more energy than glycogen storage can accommodate. Most discussions of weight gain focus on sugar consumption or eating too frequently, overlooking the dysbiosis-driven pathway. Addressing gut dysbiosis directly is described as equally important to avoiding future antibiotic exposure for resolving weight issues. The liver converts remaining excess glucose into fat after glycogen stores reach capacity. Fat storage occurs when refined carbohydrate intake is greater than what the body can immediately use. Stored fat from excess refined carbohydrates can accumulate in inactive areas of the body. Excess energy that cannot be stored as glycogen is redirected directly into fat cells. Stored fat from excess refined carbohydrates can accumulate on internal organs. Glucose absorbed in excess of what cells can immediately use or store as carbohydrate is converted into body…